Ancestral Diet: Is it the best for you?

Do you believe that you should have a diet that replicates your ancestors or one that follows current health recommendations/trends?

"A new study of hundreds of human genomes has revealed that groups in various regions of the world have evolved for diets with different amounts of meat and vegetables. People from Europe, particularly its southern regions, are optimized for a high-plant diet. But people from other areas, such as the Inuit of Greenland, have a biochemistry that is better able to process lots of meat fat."

https://arstechnica.com/science/2017...and-years-ago/
Study from 2017: https://academic.oup.com/mbe/article/34/6/1307/3062804

I have to resort to drinking Ancient Iranian Psychedelics because my dealer isn't working during the quarantine

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Originally Posted by Dr_Maul

I have to resort to drinking Ancient Iranian Psychedelics because my dealer isn't working during the quarantine

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The best indeed lol

It means I have to eat sour cream, paprika, Bogrács from a cauldron and drink koumiss my entire life?

Okay! :D

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Originally Posted by Chris596

It means I have to eat sour cream, paprika, Bogrács from a cauldron and drink koumiss my entire life?

Okay! :D

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What’s koumiss? We have kımız, are they the same drink?

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Originally Posted by itilvolga

What’s koumiss? We have kımız, are they the same drink?

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It is the same

I diet of graf/beans. Nice, but i couldn't eat it all the time.

https://blog.arousingappetites.com/w...4/IMG_8579.jpg

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Originally Posted by itilvolga

What’s koumiss? We have kımız, are they the same drink?

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I just looked up kımız. Yes, it's the same, it comes from nomadic steppe people as far as I know. This is why Turkish people and Hungarians know it :) But it's not really a common drink here I think. I have tried it before, also in Turkey. It has a sour taste. Sorry for the late reply!

Ancestral breakfast

https://i.imgur.com/W0JuotP.jpg

The LCT gene provides the instructions for making lactase. The specific DNA sequence in the MCM6 gene helps control whether the LCT gene is turned on or off. At least several thousand years ago, some humans developed a mutation in the MCM6 gene that keeps the LCT gene turned on even after breast feeding is stopped. Populations that are lactose intolerant lack this mutation. The LCT and MCM6 genes are both located on the long arm (q) of chromosome 2 in region 21. The locus can be expressed as 2q21. The lactase deficiency also could be linked to certain heritages. It is more common in Asian Americans, African Americans, Mexican Americans, and Native Americans. Analysis of the DNA of 94 ancient skeletons in Europe and Russia concluded that the mutation for lactose tolerance appeared about 4,300 years ago and spread throughout the European population.

Some human populations have developed lactase persistence, in which lactase production continues into adulthood probably as a response to the benefits of being able to digest milk from farm animals. Some have argued that this links intolerance to natural selection favoring lactase-persistent individuals, but it is also consistent with a physiological response to decrease lactase production when it is not needed in cultures in which dairy products are not an available food source. Although populations in Europe, India, Arabia, and Africa were first thought to have high rates of lactase persistence because of a single mutation, lactase persistence has been traced to a number of mutations that occurred independently. Different alleles for lactase persistence have developed at least three times in East African populations, with persistence extending from 26% in Tanzania to 88% in the Beja pastoralist population in Sudan.

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Triticeae glutens are important factors in several inflammatory diseases. The immunochemistry can be subdivided into innate responses (direct stimulation of immune system), class II mediated presentation (HLA-DQ), class I mediated stimulation of killer cells, and antibody recognition. The responses to gluten proteins and polypeptide regions differs according to the type of gluten sensitivity. The response is also dependent on the genetic makeup of the human leukocyte antigen genes. In enteropathy, there are at least 3 types of recognition, innate immunity (a form of cellular immunity priming), HLA-DQ and antibody recognition of gliadin and transglutaminase. In NCGS, there is high AGA IgG in more than half of the cases. In wheat allergy, there appears to be an innate component and the response pathways are mediated through IgE against gliadin and other wheat proteins.

Celiac disease (CD) and Non-celiac gluten sensitivity (NCGS) are closely linked with human leukocyte antigen (HLA) class II genes, HLA-DQ2 and HLA-DQ8, located on chromosome 6p21. Nearly all CD people are HLA-DQ2/HLA-DQ8 positive, with 95% HLA-DQ2 and the rest usually HLA-DQ8 (which is carried by 30% of Caucasians). However, the specificity of HLA-DQ2 and/or HLA-DQ8 for CD is low, with estimates ranging from 36% to 53%. In persons with NCGS, the HLA-DQ2 and/or HLA-DQ8 alleles are present in only about 50%, which is still a greater proportion than in the general population.