The biggest obstacle in proving this both plausible and probable theory is that these ninja-ass motherfuckers leave "no" traces behind, smooth criminal style, so every piece of evidence for this theory is indirect, meaning the answer to the biggest dilemma in biology will remain forever unsolvable. There is only the equivalent of burglars dropping a "hair" like in GATTACA (1997) and I'mma finna bouta drop them "hairs" now.

_______________

Overview:
• Philip Bell "The Viral Eukaryogenesis Theory" from Cellular Origin and Life in Extreme Habitats (2005)
"It is proposed in the viral eukaryogenesis theory that the eukaryotic nucleus is
descended from an ancient NCLDV-like virus that infected an archaeal host."
• "Assembly of a nucleus-like structure during viral replication in bacteria."
• Spooky eukaryote-like activity in the prokaryotic world: "A phage tubulin assembles dynamic filaments by an atypical mechanism to center viral DNA within the host cell."
https://www.ncbi.nlm.nih.gov/pubmed/22726436
This is the first example of a prokaryotic tubulin array that functions analogously to the microtubule-based spindles of eukaryotes
• T7 phage info "Structure of human mitochondrial RNA polymerase."
• T7 phage info: "Mechanism of transcription initiation by the yeast mitochondrial RNA polymerase."
• Most likely scenario (fig 1) (fig 2 - simplified)

Look up also:
- Review: Pennisi, Elizabeth "The Birth of the Nucleus" (2004)
- "Phage WO of Wolbachia: lambda of the endosymbiont world"
- Intracellular life cycle of Chlamydiae
"from pathogens of humans and animals to symbionts of ubiquitous protozoa"
- Parasitic nucleus of a red alga; example of all-nucleus quasi-algae Physarum polycephalum (>>>More).
- Damage to mitochondria's TWINK (T7-like protein) has a role in pathophysiology (eyesight, neurodegenerative outcome)
Mutations occurring on the TWNK gene are associated with health conditions such as Perrault Syndrome, ataxia neuropathy spectrum, infantile-onset spinocerebellar ataxia, and most prominently progressive external ophthalmoplegia.
- Evolutionary pozzin' hasn't ended yet? (syncytiotrophoblast, ERV, ERVWE1)
- Compton, Alex "They Might Be Giants, Does Syncytium Formation Sink or Spread HIV Infection" (2017)
"[T]he in vivo study of other viruses known to induce [endometrial] syncytia in vitro, such as nonlentiviral retroviruses (T lymphotropic virus and foamy virus) and RNA viruses (measles virus and respiratory syncytial virus)"
LMFAO!!! Imagine that the whole mammalian reproduction "apparatus" is carried out by HIV-like poz genes or that the "end" result of primordial ooze viruses was human because that's exactly where we at, homeboy.


Related threads:

Endosymbiotic component 1: Alphaproteobacteria ATP (energy/power) production center (mitochondria).



The mitochondrion motherfucker in the above picture, in order to survive and in order for everyone to survive, uses reproduction tools (TWINK gene codes for proteins "Twinkle" and "Twinky") as this bad boy (T7 phage) in the next slide so if the TWINK which is viral and not bacterial (alphaproteobacteria), as you would expect, in origin is damaged you literally become a twitching retard. That's fucking loco, ese.




Endosymbiotic component 2: Proto-nucleus




* * *



Sexual reproduction originates from viral replication?

Endosymbiotic component 3: In the extremophile (archeal) hood