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By Bill Sardi
In December of 2019 a lower respiratory tract (lung) illness accompanied by fever of unknown origin was reported in a cluster of patients in Wuhan, China. Because of prior outbreaks of coronavirus infections in China, coronaviruses were initially suspected and a newly mutated strain was identified that human populations had no immunity towards. This infectious disease was coined COVID-19 and is usually diagnosed by symptomology (dry cough, fatigue, muscle aches, fever, with chest x-rays revealing pneumonia (fluid in the lungs) and an accompanying ground-glass appearance in the lungs being a hallmark sign.
There are two symptoms of interest that pertain to COVID-19:
1st symptom
The hallmark, but not universal, sign – – – of COVID-19 are opacities in the lower lungs that appear like ground glass in a lung scan. These opacities represent fluid in the lung space which prevents oxygen from being transferred to hemoglobin in red blood cells, resulting in shortness of breath.
Ground-glass opacities were reported in up to 86.1% of 101 COVID-19 coronavirus cases of pneumonia. Patients with extensive ground-glass opacity (greater than 50%) are more likely to experience a longer hospital stay suffer acute respiratory failure than those with less ground-glass opacity.
Here is what ground-glass opacities look like in a lung scan (inside red boxes).
Ground glass opacities are linked to use of toxic and non-toxic drugs. More than 600 drugs known to cause lung toxicity. Many of the drugs used in the hospital to treat infectious lung diseases are toxic to the lungs! Sedatives and tranquilizers may induce pneumonia. The more medicine doctors prescribe the more likely you run into one of these hundreds of lung-toxic drugs.
The appearance of ground-glass lung opacities are far more likely among blacks who characteristically have lower vitamin D levels than Caucasians, and are less common in summer months when sunshine vitamin D levels are higher, and is 4.3 times more likely among individuals with vitamin D deficiency.
This is just one reason why hospitalization should be avoided at all cost. Another reason is to avoid treatment errors and antibiotic resistant bacteria that live in hospitals. And because hospital rooms are dark caves where sun-starvation induces vitamin D deficiency. All patients with suspected infectious lung disease should be screened for vitamin D deficiency upon hospital admission. This isn’t being done.
2nd symptom
Crackling sounds in the lungs are the second symptom of interest. By definition, cracklesare the clicking, rattling, or cracklingnoises that may be made by one or both lungs of a human with a respiratory disease during inhalation. They are heard only with a stethoscope (“auscultation”). You can hear crackles in an online video.
Crackle sounds in the lungs are not specific for any particular infectious disease but they are widely associated with…… vitamin B1 deficiency. Here is the evidence:
▪Patients with beriberi (vitamin B1 – – thiamine) deficiency are known to have crackle sounds in their lungs.
▪The World Health Organization lists lung crackles in the lower lungs as sign of thiamine deficiency. B1 deficiency is also known as beriberi.
▪ Many hospitalized patients are placed on water pills (diuretics) to control blood pressure with no regard for the fact this class of drugs deplete vitamin B1.
▪ Infections were reported among 35 of 68 patients who had pneumonia. Infection may be the presenting manifestation of vitamin B1 deficiency.
▪ Cases of pneumonia were frequently reported in prisoner of war camps when beriberi developed.
▪A 73-year old man with beriberi breathing had crackles in both lungs which resolved with vitamin B1 therapy.
▪Vitamin B1 helps to limit the growth of the bacterium that causes tuberculosis.
▪ Patients with tuberculosis are more likely to have low vitamin B1 blood levels.
▪ One published scientific report refers to thiamine (B1) as a “super antibiotic.”
https://www.karger.com/Article/Fulltext/484699
Nicotine and alcohol use deplete essential nutrients like vitamin B1 and vitamin C. In China, 68% of men are smokers and 46% drink alcohol.
What may be missed by acute care physicians is that fever with pneumonia among some patients may not be induced by a germ but may be induced by a vitamin B1 deficiency that results from loss of control of body temperature by the hypothalamus in the brain. Thiamine deficiency can result in high fever. Vitamin B1 injections may eradicate infections. A fever accompanied by vitamin B1 malnutrition may emanate from dysfunction of the hypothalamus in the brain with accompanying lack of nitric oxide, a transient gas in the blood circulation needed to quell infections.
Many coronavirus-infected patients are alcoholics. Alcohol induces vitamin B1 deficiency. In many cases of pneumonia no bacterial or viral infection is identified. Vitamin B1 therapy should be a standard therapy for any patient with lung disease and a history of alcohol or tobacco consumption should call for vitamin D testing upon hospital admission.
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